While this 1/2 typology is incomplete, diabetic ketoacidosis is a phenomenon generally relevant to type 1 family of conditions.
Normally (even with pure type 2!) glucose-ketone levels move in opposition: to put it simply ketone production is activated by low insulin, which is activated by low glucose. Ketone bodies are then consumed instead of glucose or excreted and generally homeostasis ensures that ketone levels do not approach anywhere close to ketoacidosis levels.
In cases where insulin production is broken (Type I diabetes, monogenic diabetes, mody) despite exogenous carbs raising blood glucose levels ketone body production is still active, however in most cases glucose is the preferred energy source, which leads to broken homeostasis of ketones, ketones accumulating and resulting diabetic ketoacidosis.
Type II family is characterized by high insulin. Some call it insufficient insulin, some call it insulin resistance, but the result is that insulin remains high and ketone production low. Regardless if the person is nominally healthy or has insulin resistance, the only way to trigger ketone production is to get insulin low for extended period, which is caused by lack of exogenous glucose. So either ketogenic diet or full-blown fasting.
Advanced and prolonged Type II diabetes (even if treated with insulin) can cause insulin production to drop along with insulin resistance, so the patient could develop Type I-like symptoms and risks, including DKA.
Normally (even with pure type 2!) glucose-ketone levels move in opposition: to put it simply ketone production is activated by low insulin, which is activated by low glucose. Ketone bodies are then consumed instead of glucose or excreted and generally homeostasis ensures that ketone levels do not approach anywhere close to ketoacidosis levels.
In cases where insulin production is broken (Type I diabetes, monogenic diabetes, mody) despite exogenous carbs raising blood glucose levels ketone body production is still active, however in most cases glucose is the preferred energy source, which leads to broken homeostasis of ketones, ketones accumulating and resulting diabetic ketoacidosis.
Type II family is characterized by high insulin. Some call it insufficient insulin, some call it insulin resistance, but the result is that insulin remains high and ketone production low. Regardless if the person is nominally healthy or has insulin resistance, the only way to trigger ketone production is to get insulin low for extended period, which is caused by lack of exogenous glucose. So either ketogenic diet or full-blown fasting.
Advanced and prolonged Type II diabetes (even if treated with insulin) can cause insulin production to drop along with insulin resistance, so the patient could develop Type I-like symptoms and risks, including DKA.